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Molecular Cancer Therapeutics
Molecular Cancer Therapeutics
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About the Cover

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On The Cover

Met kinase homology model (top panel). A homology model of the Met kinase was developed based on the crystal structures of the FGFR1 receptor kinase. ATP was docked into the active site based on the co-crystal of FGFR1 with ATP. Amino acids within 6 Angstroms of ATP that differ between Met and FGFR1 are highlighted in yellow. Lower panel shows the Met kinase homology model with a synthetic Met inhibitor, SU11271, docked in ATP binding site. For details, see 1085 et al. in this issue.

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Molecular Cancer Therapeutics: 2 (11)
November 2003
Volume 2, Issue 11
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Issue Highlights

  • Potent and selective inhibitors of the Met [hepatocyte growth factor/scatter factor (HGF/SF) receptor] tyrosine kinase block HGF/SF-induced tumor cell growth and invasion
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Copyright © 2021 by the American Association for Cancer Research.

Molecular Cancer Therapeutics
eISSN: 1538-8514
ISSN: 1535-7163

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