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Volume 11, Issue 2, pp. 257-266
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History
- Received July 13, 2011
- Revision received October 6, 2011
- Accepted October 12, 2011
- Published first February 8, 2012.
Copyright & Usage
©2012 American Association for Cancer Research.
Author Information
- Authors' Affiliations: 1Department of Pharmacology and Molecular Sciences, and 2Sidney Kimmel Comprehensive Cancer Center at Johns Hopkins, Johns Hopkins University, Baltimore, Maryland
- Corresponding Author:
Samuel R. Denmeade, Department of Oncology, Johns Hopkins University School of Medicine, Cancer Research Building I, Rm. 1M43, 1650 Orleans St., Baltimore MD 21231. Phone: 410-955-8875; Fax: 410-614-8397; E-mail: denmesa{at}jhmi.edu
February 2012
Volume 11, Issue 2
Volume 11, Issue 2
Rationale Behind Targeting Fibroblast Activation Protein–Expressing Carcinoma-Associated Fibroblasts as a Novel Chemotherapeutic Strategy
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- Article
- Abstract
- Introduction
- Fibroblast Activation Protein and the Post-Prolyl Peptidase Family
- FAP Expression in the Tumor Microenvironment
- Role of FAP in the Biology of Cancer
- Potential for Therapeutic Exploitation of FAP by a Novel Prodrug
- Conclusions
- Disclosure of Potential Conflicts of Interest
- Grant Support
- References
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